Norbert Perrimon, Ph.D.
James Stillman Professor of Developmental Biology, Harvard Medical School
Investigator, Howard Hughes Medical Institute
The Drosophila stubarista phenotype is associated with a dosage effect of the putative ribosome-associated protein D-p40 on spineless.
The torso receptor protein-tyrosine kinase signaling pathway: an endless story.
Simple and efficient generation of marked clones in Drosophila.
Targeted gene expression as a means of altering cell fates and generating dominant phenotypes.
Approaches to identify genes involved in Drosophila embryonic CNS development.
Authors: Authors: Noll E, Perkins LA, Mahowald AP, Perrimon N.
J Neurobiol
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J Neurobiol
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Developmental and molecular characterization of mutations in the Drosophila-raf serine/threonine protein kinase.
Authors: Authors: Melnick MB, Perkins LA, Lee M, Ambrosio L, Perrimon N.
Development
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Development
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Control of cell fate determination by p21ras/Ras1, an essential component of torso signaling in Drosophila.
Authors: Authors: Lu X, Chou TB, Williams NG, Roberts T, Perrimon N.
Genes Dev
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Genes Dev
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The torso pathway in Drosophila: a model system to study receptor tyrosine kinase signal transduction.
Cell patterning in the Drosophila segment: engrailed and wingless antigen distributions in segment polarity mutant embryos.
Authors: Authors: van den Heuvel M, Klingensmith J, Perrimon N, Nusse R.
Dev Suppl
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Dev Suppl
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wingless signaling acts through zeste-white 3, the Drosophila homolog of glycogen synthase kinase-3, to regulate engrailed and establish cell fate.