Norbert Perrimon, Ph.D.
James Stillman Professor of Developmental Biology, Harvard Medical School
Investigator, Howard Hughes Medical Institute
The Drosophila segment polarity gene dishevelled encodes a novel protein required for response to the wingless signal.
Ectopic expression in Drosophila.
Authors: Authors: Brand AH, Manoukian AS, Perrimon N.
Methods Cell Biol
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Methods Cell Biol
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Mutations in the segment polarity genes wingless and porcupine impair secretion of the wingless protein.
Authors: Authors: van den Heuvel M, Harryman-Samos C, Klingensmith J, Perrimon N, Nusse R.
EMBO J
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EMBO J
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The torso pathway in Drosophila: a model system to study receptor tyrosine kinase signal transduction.
Cell patterning in the Drosophila segment: engrailed and wingless antigen distributions in segment polarity mutant embryos.
Authors: Authors: van den Heuvel M, Klingensmith J, Perrimon N, Nusse R.
Development
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Development
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Autosomal P[ovoD1] dominant female-sterile insertions in Drosophila and their use in generating germ-line chimeras.
The Drosophila stubarista phenotype is associated with a dosage effect of the putative ribosome-associated protein D-p40 on spineless.
The torso receptor protein-tyrosine kinase signaling pathway: an endless story.
Simple and efficient generation of marked clones in Drosophila.
Targeted gene expression as a means of altering cell fates and generating dominant phenotypes.